Fault feature extraction method based on EWT-SMF and MF-DFA for valve fault of reciprocating compressor

According to the nonlinearity and nonstationarity characteristics of reciprocating compressor vibration signal, a fault feature extraction method of reciprocating compressor based on the empirical wavelet transform (EWT) and state-adaptive morphological filtering (SMF) is proposed. Firstly, an adaptive empirical wavelet transform was used to divide the Fourier spectrum by constructing a scale-space curve, and an appropriate orthogonal wavelet filter bank was constructed to extract the AM-FM component with a tightly-supported Fourier spectrum. Then according to the impact characteristic of the reciprocating compressor vibration signal, the morphological structural elements were constructed with the characteristics of the signal to perform state-adaptive morphological filtering on the partitioned modal functions. Finally, the MF-DFA method of the modal function was quantitatively analyzed and the fault identification was performed. By analyzing the experimental data, it can be shown that the method can effectively identify the fault type of reciprocating compressor valve

Anillin Regulates Neuronal Migration and Neurite Growth by Linking RhoG to the Actin Cytoskeleton

Neuronal migration and neurite growth are essential events in neural development, but it remains unclear how guidance cues are transduced through receptors to the actin cytoskeleton, which powers these processes. We report that a cytokinetic scaffold protein, Anillin, is redistributed to the leading edge of the C. elegans Q neuroblast during cell migration and neurite growth. To bypass the requirement for Anillin in cytokinesis, we used the somatic CRISPR-Cas9 technique to generate conditional mutations in Anillin. We demonstrate that Anillin regulates cell migration and growth cone extension by stabilizing the F-actin network at the leading edge. Our biochemical analysis shows that the actin-binding domain of Anillin is sufficient to stabilize F-actin by antagonizing the F-actin severing activity of Cofilin. We further uncover that the active form of RhoG/MIG-2 directly binds to Anillin and recruits it to the leading edge. Our results reveal a novel pathway in which Anillin transduces the RhoG signal to the actin cytoskeleton during neuronal migration and neurite growth